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The binding of NAPQI to mitochondrial proteins contributes to mitochondrial dysfunction (Lancaster et al., 2015) following mitochondrial GSH (mGSH) depletion, causing the generation of reactive oxygen species (ROS), oxidative stress and mitochondrial DNA damage

G., Pahima, H., Levi-Schaffer, F

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reviewed the data from pivotal clinical trials of people from different age subgroups receiving targeted cancer therapies and found that people aged 65 years or older shared similarities with their younger counterparts in therapy tolerance and efficacy

The elucidation of the lung-brain axis has profoundly transformed our understanding of the etiology of neurological disorders and opened promising new avenues for the development of cross-system intervention strategies